Aim: To determine the hypertension in COVID-19 patients. Material and Methods: In the current retrospective cohort analysis, a total of one hundred COVID-19 patients who were hospitalised to the same hospital during the course of this study's duration were included. Patients those who were younger than 18 years old, steroid medication, history of systemic inflammatory illness,history of kidney or liver failure, hypertension, left the follow-up, and who had missing data were excluded.Results: One hundred COVID-19 patients, with an average age of 45.69 ±5.66 years, were included in the study; of those patients, 65 (or 65%) were female. On admission, the serum D-dimer level was 150.50 (99.50–298.50) ng/mL, which was significantly higher than the post-COVID-19 period level of 120.25 (98.65–191.25) ng/mL. The CRP level was 5.2 (2.24–9.55) mg/L, which was significantly higher than the post-COVID-19 period level of 3.3 (2.2–5.5) mg/L. Following COVID-19, there was a statistically significant drop in high sensitivity troponin-I (10.0±2.63 pg/mL compared to 4.1±1.2 pg/mL, P<0.001). Both the systolic blood pressure (119.88±6.39 vs. 127.53±7.61 mmHg, P<0.001) and the diastolic blood pressure (77.91±5.59 vs. 82.99±8.36 mmHg, P.001) were significantly higher in the post-COVID-19 period when compared with the readings taken on admission.Conclusion: In conclusion, the findings of the current investigation demonstrated that COVID-19 generates new cases of hypertension and leads to a rise in both systolic and diastolic blood pressure. The findings of this research have important implications for clinical practise.